Zhou FD, Zhao MH, Zou WZ, Liu G, Wang H. The changing spectrum of primary glomerular diseases within 15 years: a survey of 3331 patients in a single Chinese centre. Nephrol Dial Transplant. 2009;24:870–6.
Article
Google Scholar
D’Amico G. Natural history of idiopathic IgA nephropathy: role of clinical and histological prognostic factors. Am J Kidney Dis. 2000;36:227–37.
Article
Google Scholar
Mulay SR, Evan A, Anders HJ. Molecular mechanisms of crystal-related kidney inflammation and injury. Implications for cholesterol embolism, crystalline nephropathies and kidney stone disease. Nephrol Dial Transpl. 2014;29:507–14.
Article
CAS
Google Scholar
Fu R, Guo C, Wang S, Huang Y, Jin O, Hu H, Chen J, Xu B, Zhou M, Zhao J, et al. Podocyte activation of NLRP3 inflammasomes contributes to the development of proteinuria in lupus nephritis. Arthritis Rheumatol. 2017;69:1636–46.
Article
CAS
Google Scholar
Shigeoka AA, Mueller JL, Kambo A, Mathison JC, King AJ, Hall WF, Correia Jda S, Ulevitch RJ, Hoffman HM, McKay DB. An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury. J Immunol. 2010;185:6277–85.
Article
CAS
Google Scholar
Xiong J, Wang Y, Shao N, Gao P, Tang H, Su H, Zhang C, Meng XF. The expression and significance of NLRP3 inflammasome in patients with primary glomerular diseases. Kidney Blood Press Res. 2015;40:344–54.
Article
CAS
Google Scholar
Li G, Chen Z, Bhat QM, Zhang Q, Abais-Battad JM, Conley SM, Ritter JK, Li PL. NLRP3 inflammasome as a novel target for docosahexaenoic acid metabolites to abrogate glomerular injury. J Lipid Res. 2017;58:1080–90.
Article
CAS
Google Scholar
Menon MC, Chuang PY, He JC. Role of podocyte injury in IgA nephropathy. Contrib Nephrol. 2013;181:41–51.
Article
Google Scholar
Goldwich A, Burkard M, Olke M, Daniel C, Amann K, Hugo C, Kurts C, Steinkasserer A, Gessner A. Podocytes are non hematopoietic professional antigen-presenting cells. J Am Soc Nephrol. 2013;24:906–16.
Article
CAS
Google Scholar
Wang C, Ye Z, Peng H, Tang H, Liu X, Chen Z, Yu X, Lou T. Effect of aggregated immunoglobulin A1 from immunoglobulin A nephropathy patients on nephrin expression in podocytes. Nephrology (Carlton). 2009;14:213–8.
Article
CAS
Google Scholar
Wang C, Peng H, Tang H, Liu X, Chen Z, Lou T, Yu X. Serum IgA1 from IgA nephropathy patients induces apoptosis in podocytes through direct and indirect pathways. Clin Invest Med. 2007;30:E240–9.
Article
CAS
Google Scholar
Yang L, Zhang X, Peng W, Wei M, Qin W. MicroRNA-155-induced T lymphocyte subgroup drifting in IgA nephropathy. Int Urol Nephrol. 2017;49:353–61.
Article
CAS
Google Scholar
Huang X, Xue H, Ma J, Zhang Y, Zhang J, Liu Y, Qin X, Sun C. Salidroside ameliorates Adriamycin nephropathy in mice by inhibiting beta-catenin activity. J Cell Mol Med. 2019;23:4443–53.
Article
CAS
Google Scholar
Ji L, Chen X, Zhong X, Li Z, Yang L, Fan J, Tang W, Qin W. Astragalus membranaceus up-regulate Cosmc expression and reverse IgA dys-glycosylation in IgA nephropathy. BMC Compl Altern Med. 2014;14:195.
Article
Google Scholar
Zhao J, Rui HL, Yang M, Sun LJ, Dong HR, Cheng H. CD36-mediated lipid accumulation and activation of NLRP3 inflammasome lead to podocyte injury in obesity-related glomerulopathy. Mediat Inflamm. 2019;2019:3172647.
Google Scholar
Gao P, He FF, Tang H, Lei CT, Chen S, Meng XF, Su H, Zhang C. NADPH oxidase-induced NALP3 inflammasome activation is driven by thioredoxin-interacting protein which contributes to podocyte injury in hyperglycemia. J Diabetes Res. 2015;2015:504761.
PubMed
PubMed Central
Google Scholar
Zhang C, Boini KM, Xia M, Abais JM, Li X, Liu Q, Li PL. Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia. Hypertension. 2012;60:154–62.
Article
CAS
Google Scholar
Schroder K, Tschopp J. The inflammasomes. Cell. 2010;140:821–32.
Article
CAS
Google Scholar
Chun J, Chung H, Wang X, Barry R, Taheri ZM, Platnich JM, Ahmed SB, Trpkov K, Hemmelgarn B, Benediktsson H, et al. NLRP3 localizes to the tubular epithelium in human kidney and correlates with outcome in IgA nephropathy. Sci Rep. 2016;6:24667.
Article
CAS
Google Scholar
Wang W, Wang X, Chun J, Vilaysane A, Clark S, French G, Bracey NA, Trpkov K, Bonni S, Duff HJ, et al. Inflammasome-independent NLRP3 augments TGF-beta signaling in kidney epithelium. J Immunol. 2013;190:1239–49.
Article
CAS
Google Scholar
Banas MC, Banas B, Hudkins KL, Wietecha TA, Iyoda M, Bock E, Hauser P, Pippin JW, Shankland SJ, Smith KD, et al. TLR4 links podocytes with the innate immune system to mediate glomerular injury. J Am Soc Nephrol. 2008;19:704–13.
Article
CAS
Google Scholar
Lorenz G, Darisipudi MN, Anders HJ. Canonical and non-canonical effects of the NLRP3 inflammasome in kidney inflammation and fibrosis. Nephrol Dial Transplant. 2014;29:41–8.
Article
CAS
Google Scholar
Anders HJ, Suarez-Alvarez B, Grigorescu M, Foresto-Neto O, Steiger S, Desai J, Marschner JA, Honarpisheh M, Shi C, Jordan J, et al. The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1-mediated tissue injury. Kidney Int. 2018;93:656–69.
Article
CAS
Google Scholar