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Correction to: Nerve growth factor improves functional recovery by inhibiting endoplasmic reticulum stress-induced neuronal apoptosis in rats with spinal cord injury

The Original Article was published on 15 May 2014

Correction to: J Transl Med 12:130 (2014) https://doi.org/10.1186/1479-5876-12-130

The original publication of this article [1] contained 4 errors in Figs. 1a, 5a, 7b, c. The wrong figures were used for the experimental animals and experimental groups. The correct figures are published in this correction article along with an updated caption. The full figures and captions can be accessed via the original article.

Fig. 1
figure 1

A. HE staining results of SCI rat at 1, 3, 7 and 14 d after contusion

Fig. 5
figure 5

A. HE staining results of the sham, SCI group and SCI rat treated with NGF group

Fig. 7
figure 7

B. The protein expressions of GAP43 in sham, SCI rats and SCI rats treated with NGF groups. GAPDH was used as the loading control and for band density normalization. C. The optical density analysis of GAP43 protein. ** P < 0.01 versus the SCI group, and ### represents P < 0.005 versus the sham group. Data are the mean values ± SEM, n = 6

The effect of the nerve growth factor (NGF) group in promoting damage repair has not changed and the changes do not affect the scientific significance of current article.

Reference

  1. Zhang H, Wu F, Kong X, Yang J, Chen H, Deng L, Cheng Y, Ye L, Zhu S, Zhang X, Wang Z, Shi H, Fu X, Li X, Xu H, Lin L, Xiao J. Nerve growth factor improves functional recovery by inhibiting endoplasmic reticulum stress-induced neuronal apoptosis in rats with spinal cord injury. J Transl Med. 2014;12:130. https://doi.org/10.1186/1479-5876-12-130.

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Correspondence to Li Lin or Jian Xiao.

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Zhang, H., Wu, F., Kong, X. et al. Correction to: Nerve growth factor improves functional recovery by inhibiting endoplasmic reticulum stress-induced neuronal apoptosis in rats with spinal cord injury. J Transl Med 19, 445 (2021). https://doi.org/10.1186/s12967-021-02901-7

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  • DOI: https://doi.org/10.1186/s12967-021-02901-7