Open Access

Erratum to: Oncolytic virus efficiency inhibited growth of tumour cells with multiple drug resistant phenotype in vivo and in vitro

  • Elena P. Goncharova1,
  • Julia S. Ruzhenkova1,
  • Ivan S. Petrov1, 3,
  • Sergey N. Shchelkunov2 and
  • Marina A. Zenkova1Email author
Journal of Translational Medicine201614:280

https://doi.org/10.1186/s12967-016-1041-3

Published: 27 September 2016

The original article was published in Journal of Translational Medicine 2016 14:241

Erratum to: J Transl Med (2016) 14:241 DOI 10.1186/s12967-016-1002-x

Unfortunately, the original version of this article [1] contained an error. Figures 2 and 7 were the incorrect versions. They have been corrected in the original article and are also included correctly in this erratum.
Fig. 2

Development of LIVP-GFP infection in various tumour cells. Development of LIVP-GFP infection in RLS (green circles), RLS-40 (red circles), KB-3-1 (violet squares), KB-8-5 (black line with open squares) and melanoma B-16 (blue triangles) tumour cells at MOI of 1 (a) and MOI of 10 (b). Cells were incubated with virus for 1 h, washed with PBS and incubated up to the analysis in IMDM supplemented with 2 % FBS. Viral titre was measured by PFU assay. Data of three independent experiments are presented

Fig. 7

Effect of LIVP-GFP treatment on the immune responses of tumour-bearing mice. a Summary data showing a significant increase of the number of IFN-γ-ransecreting splenocytes in LIVP-GFP-treated mice (n = 6) with RLS-40 or with melanoma B-16 tumours. Comparison of immune-related proteins GMC-SF (b) and IL-6 (c) in the blood serum of RLS-40 bearing mice during the treatment with LIVP-GFP (the experimental scheme was shown in Fig. 5a): red circles and blue squares for RLS-40 bearing mice treated with LIVP-GFP and PBS, respectively; black triangles healthy mice receiving PBS. The levels of cytokines in the blood serum were measured by ELISA. For each day, the value of mean ± SEM is shown

Notes

Declarations

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors’ Affiliations

(1)
Institute of Chemical Biology and Fundamental Medicine SB RAS
(2)
Institute of Cytology and Genetics SB RAS
(3)
Department of Biochemistry, Biocenter, University of Wuerzburg

Reference

  1. Goncharova EP, Ruzhenkova JS, Petrov IS, Shchelkunov SN, Zenkova MA. J Transl Med. 2016;14:241. doi:10.1186/s12967-016-1002-x.View ArticlePubMedPubMed CentralGoogle Scholar

Copyright

© The Author(s) 2016

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