Fig. 8From: Pretreatment of UC-MSCs with IFN-α2 improves treatment of liver fibrosis by recruiting neutrophilsPotential mechanism of pretreated UC-MSCs with IFN-α2 to alleviate fibrosis. IFN-α2 induced UC-MSCs to secret more IL-8, CSF-3, and CCL20 through activating the p-STAT1 and p-STAT2 pathway. In vivo treatment, pretreated UC-MSCs recruited neutrophils to play a role, and the latter is the main source of MMP8Back to article page