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Table 1 Anthropometric and biochemical variables in the non-obese and morbidly obese subjects classified according to their insulin resistance levels

From: Hypoxia is associated with a lower expression of genes involved in lipogenesis in visceral adipose tissue

  Non-obese subjects
(N = 15)
MO-low-IR
(N = 15)
MO-high-IR
(N = 15)
Sex (male/female) 4/11 5/10 5/10
Age (years) 40.3 ± 11.8 40.6 ± 11.4 37.2 ± 9.4
Weight (kg) 63.1 ± 8.5 153.6 ± 29.5a 158.7 ± 23.0a
BMI (kg/m2) 22.5 ± 1.7 55.6 ± 7.2a 57.6 ± 4.8a
Waist (cm) 80.0 ± 8.7 140.8 ± 20.3a 147.5 ± 19.6a
Hip (cm) 91.3 ± 7.7 160.1 ± 15.8a 158.1 ± 13.8a
Glucose (mg/dl) 83.1 ± 10.7 91.1 ± 10.5a 107.9 ± 15.8a*
Cholesterol (mg/dl) 192.0 ± 42.6 201.4 ± 40.4 193.5 ± 31.3
Triglycerides (mg/dl) 89.6 ± 44.9 114.7 ± 55.4 163.7 ± 77.8a*
Free fatty acids (mmol/L) 0.305 ± 0.030 0.585 ± 0.066a 0.584 ± 0.042a
Insulin (μIU/ml) 5.9 ± 3.6 12.6 ± 4.1a 39.6 ± 10.4a*
HOMA-IR 1.2 ± 0.72 2.8 ± 0.81a 10.6 ± 2.4a*
Adiponectin (μg/mL) 29.8 ± 18.5 10.9 ± 4.0a 8.0 ± 4.7a
Leptin (ng/mL) 8.2 ± 8.03 77.6 ± 47.8a 59.8 ± 21.5a
  1. The results are given as the mean ± standard deviation
  2. MO-low-IR morbidly obese subjects with low insulin resistance, MO-high-IR morbidly obese subjects with high insulin resistance, BMI body mass index, HOMA-IR homeostasis model assessment of insulin resistance index
  3. aP < 0.05 significant differences with respect to the non-obese group
  4. * P < 0.05 significant differences between morbidly obese patients with low and high insulin resistance. All significant differences are adjusted for sex