Fig. 3From: VEGF111b, a C-terminal splice variant of VEGF-A and induced by mitomycin C, inhibits ovarian cancer growthVEGF111b overexpression induces cell cycle arrest. (a) Detection of the proportion of ovarian cancer cells in the cell cycle stages after VEGF111b and VEGF165b is overexpressed, respectively. (b) The proportion of cells in the G0-G1 phase of the cell cycle decreased and the proportion of cells in the S phase increased in VEGF111b and VEGF165 group (*p < 0.05; **p < 0.01; ***p < 0.001). Bar heights represent average of data and bars represent the SEM of data obtained in three independent experimentsBack to article page