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Table 1 Clinical and pathological findings of the patients

From: Increased expression of stathmin and elongation factor 1α in precancerous nodules with telomere dysfunction in hepatitis B viral cirrhotic patients

Case no Age Sex Etiology Pathological diagnosisa
1 44 F HBV LC, LGDN, HGDN, HGDN, HCC (2)
2 52 M HBV LC, LGDN, HGDN, HGDN, HGDN, HGDN, HCC (2), HCC (2)
3 60 F HBV LC, HGDN, HGDN, HCC (1)
4 48 M HBV LC, HGDN, HGDN, HCC (1)
5 49 M HBV LC, LGDN, LGDN, LGDN, LGDN, LGDN, LGDN, HCC (3)
6 53 F HBV LGDN, HGDN
7 53 M HBV LGDN, HGDN
8 45 M HBV LC, HGDN
9 60 M HBV LC, HGDN
10 59 M HBV LC, HGDN
11 32 F HBV LC, LGDN
12 48 M HBV LC, LGDN, LGDN
13 59 M HBV LC, HCC (1),
14 56 M HBV HCC (3)
15 41 M HBV HCC (3)
16 43 M HBV HCC (3)
17 40 M HBV HCC (2)
18 60 M HBV HCC (2)
19 54 F HBV HCC (2)
20 49 M HBV HCC (2)
21 51 M HBV LGDN
22 63 M HBV HGDN, HGDN
23 48 M HBV LC
24 48 M HBV LC
  1. M, male; F, female; HBV, hepatitis B virus; LGDN, low-grade dysplastic nodule; HGDN, high-grade dysplastic nodule; HCC, hepatocellular carcinoma.
  2. aHCC grading according to the Edmondson and Steiner classification is in parentheses.