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Table 1 Clinical and pathological findings of the patients

From: Increased expression of stathmin and elongation factor 1α in precancerous nodules with telomere dysfunction in hepatitis B viral cirrhotic patients

Case no

Age

Sex

Etiology

Pathological diagnosisa

1

44

F

HBV

LC, LGDN, HGDN, HGDN, HCC (2)

2

52

M

HBV

LC, LGDN, HGDN, HGDN, HGDN, HGDN, HCC (2), HCC (2)

3

60

F

HBV

LC, HGDN, HGDN, HCC (1)

4

48

M

HBV

LC, HGDN, HGDN, HCC (1)

5

49

M

HBV

LC, LGDN, LGDN, LGDN, LGDN, LGDN, LGDN, HCC (3)

6

53

F

HBV

LGDN, HGDN

7

53

M

HBV

LGDN, HGDN

8

45

M

HBV

LC, HGDN

9

60

M

HBV

LC, HGDN

10

59

M

HBV

LC, HGDN

11

32

F

HBV

LC, LGDN

12

48

M

HBV

LC, LGDN, LGDN

13

59

M

HBV

LC, HCC (1),

14

56

M

HBV

HCC (3)

15

41

M

HBV

HCC (3)

16

43

M

HBV

HCC (3)

17

40

M

HBV

HCC (2)

18

60

M

HBV

HCC (2)

19

54

F

HBV

HCC (2)

20

49

M

HBV

HCC (2)

21

51

M

HBV

LGDN

22

63

M

HBV

HGDN, HGDN

23

48

M

HBV

LC

24

48

M

HBV

LC

  1. M, male; F, female; HBV, hepatitis B virus; LGDN, low-grade dysplastic nodule; HGDN, high-grade dysplastic nodule; HCC, hepatocellular carcinoma.
  2. aHCC grading according to the Edmondson and Steiner classification is in parentheses.