Fig. 5From: Contribution of TLR4 signaling in intermittent hypoxia-mediated atherosclerosis progressionGlycolipid disorder in vivo. a–e Total cholesterol (n = 6), high-density lipoprotein cholesterol (HDL-C) (n = 6), low-density lipoprotein cholesterol (LDL-C) (n = 6), triglycerides (TG) (n = 6) and blood glucose (n = 6) in ApoE−/− mice. *p < 0.05 versus Sham group and #p < 0.05 versus IH + LV-EGFP groupBack to article page