Fig. 9From: Integrated multi-omics analyses and functional validation reveal TTK as a novel EMT activator for endometrial cancerIllustration demonstrating the role of the TTK/miR-21 pathway in facilitating EMT and conferring chemoresistance in EC. Through a screening process, TTK was discovered to be a potential biomarker and therapeutic target for EC. Higher TTK expression in EC cells leads to increased miR-21 expression, which induces EMT, cell proliferation, and chemoresistanceBack to article page