Fig. 8From: Ca2+ dysregulation in cardiac stromal cells sustains fibro-adipose remodeling in Arrhythmogenic Cardiomyopathy and can be modulated by flecainideCharacterization and targeting of Ca2+ imbalance and related phenotypes in ACM C-MSC. Left panel: Ca2+ handling machinery and CaMKII activity in HC (top) and ACM (bottom) C-MSC. Right panel: overview of approaches used to modulate fibro-fatty accumulation by targeting of Ca2+ oscillations and CaMKII activity in ACM C-MSCBack to article page