Fig. 7From: Celastrol elicits antitumor effects by inhibiting the STAT3 pathway through ROS accumulation in non-small cell lung cancerMechanism of celastrol against NSCLC. Celastrol activated mitochondrial ROS to trigger endoplasmic reticulum stress and inhibited IL-6/STAT3 pathway to induce apoptosis in NSCLCBack to article page