Fig. 8

Graphical summary of the regulatory mechanism of LINC00665 in NSCLC radiosensitivity. LINC00665 acted as a sponge of miR-582-5p to inhibit miR-582-5p expression, up-regulated the expression of UCHL3, and enhanced the stability of AhR protein, thereby promoting the immune escape and reducing the radiosensitivity in NSCLC cells