Fig. 2From: The Apoe−/− mouse model: a suitable model to study cardiovascular and respiratory diseases in the context of cigarette smoke exposure and harm reductionLipid imbalance together with endothelial dysfunction and systemic inflammation determine the development of atherosclerosis as well as lung inflammation in Apoe−/− mice (and humans). CS exposure enhances these processes, thereby increasing oxidative stress and inflammation processesBack to article page