Figure 9From: Conditioned media from adipose stromal cells limit lipopolysaccharide-induced lung injury, endothelial hyperpermeability and apoptosis hASC-CM preserves microscopic HPAEC monolayer integrity. Preincubation of endothelial monolayers with hASC-CM suppressed H2O2-induced rearrangement of F-actin cytoskeleton and disintegration of peripheral adherens junctions. Arrows indicate the gaps appeared in previously intact monolayer in response to H2O2.Back to article page