Regulatory networks of miRNAs and PI3K/AKT pathway in controlling EMT and cancer stemness in human tumors including endometrial cancer. Activation of PI3K/AKT signaling plays an essential role in promoting EMT and cancer stem cell phenotypes. Interactions between PI3K/AKT and other pathways regulate EMT/CSC. MiRNAs function as both upstream mediators and downstream effectors to affect PI3K/AKT pathway activities. References showing the regulatory interactions are indicated. Verified interactions in endometrial cancer were underlined.